继发性甲状旁腺功能亢进 - 百度百科
网页继发性 甲状旁腺功能亢进 （SHPT，简称继发性甲旁亢），是指在 慢性肾功能不全 、肠吸收不良综合征、 Fanconi综合征 和肾小管 酸中毒 、 维生素D 缺乏或抵抗以及妊娠、哺乳等情况下，甲状旁腺长期受到 低血钙 、低血镁或高血磷的刺激而分泌过量的 PTH，以提高 血钙 、血镁和降低血磷的一种慢性代偿性临床表现，长期的 甲状旁腺增生 最终导致形成功能自主的 腺瘤。 外文名. secondary hyperparathyroidism. 就诊科室. 内科. 常见病因. 肾脏病变； …
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继发性甲状旁腺功能亢进症 - 症状、诊断和治疗 | BMJ Best Practice
网页2024年8月27日 · 继发性甲状旁腺功能亢进（secondary hyperparathyroidism, SHPT）是继发于低钙血症的甲状旁腺素（parathyroid hormone, PTH）升高。. PTH 通过作用于肾小管以及骨骼中钙库，并通过维生素 D 活化作用和肠吸收间接作用于胃肠道，从而维持钙稳态。. 与 SHPT 最常相关的疾病是慢性 ...
Secondary Hyperparathyroidism - StatPearls - NCBI Bookshelf
网页2020年8月28日 · Secondary hyperparathyroidism (SHPT) is an increased secretion of PTH due to parathyroid hyperplasia caused by triggers such as hypocalcemia, hyperphosphatemia, or decreased active vitamin D. The increased PTH secretion, in turn, causes increased calcium in the blood by acting on bones, intestines, and kidneys.
Secondary hyperparathyroidism - Wikipedia
网页Endocrinology. Secondary hyperparathyroidism is the medical condition of excessive secretion of parathyroid hormone (PTH) by the parathyroid glands in response to hypocalcemia (low blood calcium levels), with resultant hyperplasia of these glands. This disorder is primarily seen in patients with chronic kidney failure.
Secondary Hyperparathyroidism: Pathogenesis and Latest Treatment
网页The classic pathogenesis of secondary hyperparathyroidism (SHPT) began with the trade-off hypothesis based on parathyroid hormone hypersecretion brought about by renal failure resulting from a physiological response to correct metabolic disorder of calcium, phosphorus, and vitamin D.
捷氢科技 - shpt.com
网页SHPT has systematically mastered R&D capability and complete self-owned intellectual properties of stack key components, stack integration, fuel cell system integration and fuel cell propulsion system integration.
Treatment of secondary hyperparathyroidism: the clinical utility …
网页2017年6月1日 · Secondary hyperparathyroidism (SHPT), a very frequent, severe, and worsening complication of chronic kidney disease, is characterized by high serum parathyroid hormone (PTH), parathyroid gland hyperplasia, and disturbances in mineral metabolism. Clinically, SHPT shows renal osteodystrophy, vascular calcification, cardiovascular damage, and ...
Treatment of Hyperparathyroidism (SHPT) - PMC - National …
网页With the progression of SHPT, there is a histological modification of the parathyroid glands. Diffuse hyperplasia becomes nodular, with decreased expression of vitamin D receptors (VDR) and Ca-sensing receptor (CaR), which may result in …
Current treatment options for secondary hyperparathyroidism in …
网页Vitamin D system is altered since early CKD, and vitamin D deficiency is an established trigger of SHPT. Although untreated SHPT may degenerate into tertiary hyperparathyroidism with detrimental consequences in advanced CKD, best treatments for counteracting SHPT from stage 3 CKD are still debated.
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