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Science子刊:中山大学廖雯婷团队等揭示KRAS突变驱动结直肠癌免疫 ...
该研究揭示了癌基因KRAS破坏 结直肠癌 细胞内源性dsRNA稳态,扰乱“病毒拟态”效应,从而推动结直肠癌的 免疫 逃逸机制。这一发现为克服结直肠癌免疫治疗耐药性提供了新的思路。
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